红斑症

  • 网络erythroplakia
红斑症红斑症
  1. 结节性红斑症的组织病理学

    Histopathologic spectrum of erythema nodosum

  2. 目的探讨兜甲蛋白基因(LOR)在进行性对称性红斑角化症(PSEK)发病中的作用。

    Objective To investigate the causative role of the loricrin gene for PSEK .

  3. 目的:研究原发性红斑肢痛症一家系成员中SCN9A基因的突变情况。

    Objective : To detect gene mutations in a Chinese family with primary erythermalgia .

  4. 目的:了解中国汉族人进行性对称性红斑角化症(PSEK)的临床表型和遗传学特点。

    Objective : To characterize the clinical and genetic features of progressive symmetric erythrokeratoderma ( PSEK ) in 42 case from 6 Chinese Han families .

  5. 进行性对称性红斑角化症的临床表型分析和突变筛查

    Progressive Symmetric Erythrokeratodermia ( PSEK ): Phenotype Analysis and Mutation Detection

  6. 1例化学性腰交感神经切除术治疗红斑肢痛症护理

    Nursing care of a case of acromelalgia patient treated with chemical lumbar sympathectomy

  7. 中国汉族人6家系42例进行性对称性红斑角化症的临床和遗传特点分析

    Clinical and genetic features of progressive symmetric erythrokeratoderma in 42 cases from 6 Chinese Han families

  8. 一原发性红斑肢痛症家系致病基因的定位研究

    The Positioning Study of the Pathogenic Gene Loci in a Family with Autosomal Dominant Primary Erythermalgia

  9. 原发性红斑肢痛症的组织病理学表现无特异性:特殊研究显示小神经纤维密度降低

    Histopathologic findings in primary erythromelalgia are nonspecific : Special studies show a decrease in small nerve fiber density

  10. 非典型性原发性红斑性肢痛症20例的临床与治疗

    Clinical features and treatment of 20 cases of primary atypical erythromelalgia

  11. 可变性红斑角皮症1例

    Angle . Erythrokeratodermia variabilis : a case report

  12. 红斑性肢痛症肢体血流量及血液凝固性的变化

    Alterations of Blood Fiow and Coagulation in Erythermalgia

  13. 进行性对称性红斑角皮症1例

    A case of progressive symmetric erythrokeratodermia

  14. 结论:中西医结合疗法是目前治疗红斑性肢痛症较好的方法之一。

    Conclusion The combined treatment may be one of the most effective methods for treating erythromelagia .

  15. 目的研究伴发于真性红细胞增多症的红斑性肢痛症发病机制、临床表现、病理生理特点。

    Objective To study the mechanism , clinical manifestations and pathophysiological characteristics of erythromelalgia accompanying polycythemia vera .

  16. 血栓形成是红斑性肢痛症各种临床表现的主要因素,应尽可能做到早期诊断,早期运用抗血小板药物治疗,发展至终末期治疗效果不佳。

    Formation of thrombosis is the major factor for the different clinical manifestations of erythromelalgia , which should be early diagnosed and treated with anti-platelet drugs .

  17. 2004年一种痛性皮肤病原发性红斑性肢痛症使人们认识到钠离子通道表达的增强是疼痛的重要原因,神经上与致痛有关的离子通道和疼痛的发生密切相关。

    In 2004 , people knew the increase of sodium channel expression was a import reason of pain through primary erythermalgia . Ion channels which trigger pain is tightly relative to the occurrence of pain .