溶血卵磷脂

rónɡ xuè luǎn lín zhī
  • lysolecithin;lysophosphatidyl choline
溶血卵磷脂溶血卵磷脂
  1. 目的探讨溶血卵磷脂(LPC)在胰性脑病(PE)发病机制中的作用环节和效应机制。

    Objective To explore some certain processes and effects of lysolecithin choline ( LPC ) in the pathogenesis of pancreatic encephalopathy ( PE ) .

  2. 溶血卵磷脂就其融合性来说,可能是研究得最为彻底的。

    Lysolecithin is probably best studied with regard to its fusing capabilities .

  3. HPLC法测定盐酸伊立替康脂质体注射液中的溶血卵磷脂

    Determination of Myristoyl in Irinotecan Hydrochloride Liposome Injection by HPLC

  4. 溶血卵磷脂对载脂蛋白E基因缺陷小鼠腹腔巨噬细胞源泡沫细胞胆固醇外流的影响

    Effects of Lysophosphatidylcholine on Cholesterol Efflux from Apolipoprotein E Gene Deficient Mouse Peritoneal Macrophage-Drived Foam Cells

  5. 以载脂蛋白E基因缺陷小鼠腹腔巨噬细胞源泡沫细胞为研究对象,观察溶血卵磷脂对泡沫细胞胆固醇外流的影响以及初步探讨其机制。

    Aim To explore the effects of lysophosphatidylcholine ( LPC ) on cholesterol efflux from apolipoprotein E ( apo E ) gene deficient mouse peritoneal macrophage foam cells .

  6. 结论:①溶血卵磷脂能引起食管黏膜损伤,当与HCL协同作用于食道黏膜时损伤作用更为显著。

    Conclusion : ( 1 ) Lysolecithin can injury the esophageal mucosa . The combined effect of lysolecithin and acid was more damaging than that of lysolecithin alone .

  7. 作者以体外培养的小牛主动脉内皮细胞为材料,研究了溶血卵磷脂胆碱(LPC)和氧自由基(OFR)对血管内皮细胞(VEC)的损伤,及儿茶素对VEC的保护。

    Effects of catechin on cultured bovine endothelial cells damaged by lysophosphatidylcholine ( LPC ) or oxygen free radicals ( OFR ) were investigated .

  8. 应用传代培养的小牛主动脉内皮细胞(BAEC)为实验模型,观察银杏叶提取物(EGb)对溶血卵磷脂胆碱(LPC)损伤BAEC及其分泌功能的影响。

    Effects of EGb on cultured bovine aortic endothelial cells ( BAEC ) damaged by lysophosphatidylcholine ( LPC ) were investigated .

  9. 溶血卵磷脂(LPC)在1、3、5、7hr明显高于NS组(P<0.05),呈进行性升高趋势。

    Lysophosphatidylcholine ( LPC ) at 1 、 3 、 5 、 7hr were higher than the NS group ( P < 0.05 ) , and increased progressively .

  10. 目的:探讨溶血卵磷脂(LPC)对巨噬泡沫细胞胆固醇外流的影响,为动脉粥样硬化(AS)的防治研究提供理论依据。

    AIM : To explore the effect of lysophosphatidylcholine ( LPC ) on cholesterol efflux from macrophage foam cells , and to offer experimental evidence for research on prevention and treatment of atherosclerosis .

  11. 目的:探讨溶血卵磷脂(LPC)对牛视网膜微血管内皮细胞(BRECs)凋亡及细胞内钙离子浓度的影响。

    Objective : To study whether lysophosphatidylcholine ( LPC ) can induce apoptosis of bovine retinal endothelial cells ( BRECs ) in vitro .

  12. 急性缺氧时,肺泡冲洗液中溶血卵磷脂、神经鞘磷脂、磷脂酰乙醇胺及磷脂酰甘油均下降(p0.05);间断适应性缺氧后,基本回复到缺氧前水平。

    On analysis of phospho-lipids in pulmonary surfactant of rabbits exposed to different hypoxic conditions ( simulated altitude 5 000m ), it was found that lysophosphatidylcholine , sphingomyelin , phosphatidylethanolamine and phosphatidylglycerol were decreased ( p0.05 ) by acute hypoxia and raised by adaptive hypoxia in lung lavage .

  13. 钙离子信使通路在溶血卵磷脂促进巨噬泡沫细胞胆固醇外流中的作用

    Effect of Calcium Signaling Pathway on Cholesterol Efflux Promoted by Lysophosphatidylcholine from Macrophage Foam Cells

  14. 溶血卵磷脂对牛视网膜微血管内皮细胞凋亡及细胞内钙离子浓度的影响

    The Effect of Lysophosphatidylcholine on the Apoptosis of Bovine Retinal Endothelial Cells and Intracellular Calcium Concentration

  15. 氧化低密度脂蛋白及溶血卵磷脂对巨噬泡沫细胞胆固醇外流的影响

    Effects of oxidized low density lipoprotein and lysophosphatidylcholine on cholesterol efflux from mouse peritoneal macrophage foam cells

  16. 具有表面活性的脂类物质中,溶血卵磷脂就其融合性来说可能是研究得最为彻底的。

    Among surface-active lipoidal substances , lysolecithin is probably the best studied with regard to its fusing capabilities .

  17. 采用磷脂酶A水解蛋黄中的卵磷脂生成溶血卵磷脂,转化度为80%。

    The lecithin was hydrolyzed with phospholipase A into the lysolecithin and the degree of conversion was 80 % .

  18. 探讨溶血卵磷脂对巨噬泡沫细胞胆固醇外流影响及第二信使细胞内钙在这一效应中所起的作用,为动脉粥样硬化的有效防治提供理论依据。

    Aim To explore the effect of lysophosphatidylcholine on cholesterol efflux from macrophage foam cells , and the function of calcium signaling pathway on this effect .

  19. 乳脂的外吐,可能与它溶血卵磷脂含量有关。

    The exocytosis of milk fat may be related to its high lysolecithin content . The patient presented with irritable crying , bilious vomiting and frank bloody stool on the 26th day of life .

  20. 结果吸收促进剂能提高胰岛素在肠道内的吸收水平,促渗强弱顺序为:1%Na2EDTA>1%水杨酸钠>1%癸酸钠>1%甘氨去氧胆酸钠>1%溶血卵磷脂;

    RESULTS All absorption enhancers promoted the hypoglycemic effect of insulin at all sites . The promotion degree was in the order of 1 % Na2 EDTA > 1 % sodium salicylate > 1 % sodium caprate > 1 % sodium glycodeoxycholate > 1 % lysolecithin .