tscc

The research on centromere protein H promoting TSCC cells proliferation

着丝粒蛋白-H对舌鳞癌细胞增殖促进作用的研究

Regulation of microRNA-21 ASO on proliferation and apoptosis of TSCC cell lines

微小RNA-21反义寡核苷酸对舌鳞癌细胞增殖和凋亡的调控作用

Objective To explore the width of safe surgical margin of TSCC by molecular biological testing .

目的通过分子生物学检测，探讨舌鳞状细胞癌（简称鳞癌）黏膜安全的切除缘宽度。

Experimental studies on the effects of tscp , TSCC on blood platelet function and their mechanism

荫风轮、风轮菜总皂甙对血小板功能影响及机理的研究

High density - low temperature combustion proposes time scale control of the combustion process ( TSCC ) concept based on chemical processes from the mixing time scale control .

高密度&低温燃烧技术是从混合与化学过程时间尺度控制的基本概念出发，提出了时间尺度控制燃烧过程（TSCC）的概念。

Results Compared to tongue normal mucosa and tongue inflammation , the expression of COX-2 was significantly higher in TSCC ( P < 0.01 ) .

结果舌鳞癌中COX2的表达明显高于正常舌黏膜和炎症组织（P<001）。

CONCLUSIONS : The positive rates of VEGF-C and Flt-4 are significantly higher in TSCC than in benign tongue epithelial lesions .

结论：VEGF-C和Flt-4在舌鳞癌的阳性率显著高于舌良性上皮性病变。

The over-expression of C-Met protein had positive correlation with clinical stage , histological differentiation and lymph node metastasis of TSCC ( P0.05 ) .

C-Met蛋白高表达与TNM分期、组织学分化及有无淋巴结转移有关（P0.05）。

Clinical score : By treatment , the scores of TSCC and symptom had decrease among three groups . The decreased of the new treatment group much lower than the other groups .

临床评分：经治疗，三组Toronto积分、症状积分均较治疗前下降，新治疗组较另外两组下降幅度大。

Objective To reveal the relations between expression of cyclooxygenase-2 ( COX-2 ) and tumor biological behavior and implications in tongue squamous cell carcinoma ( TSCC ) .

目的探讨环氧化酶2（COX2）的表达与舌鳞癌生物学行为的关系及意义。

Results : PCR-SSCP results showed that the mutation rate was 59.4 % ( 19 / 32 ) in TSCC and 20.0 % ( 2 / 10 ) in tongue leukoplakia .

结果：PCR-SSCP检测舌白斑和舌癌p53基因突变率分别为20.0%（2/10）和59.4%（19/32）。

This study was to investigate the expression and clinical significance of VEGF-C and its receptor fms-like tyrosine kinase-4 ( Flt-4 ) in tongue squamous cell carcinoma ( TSCC ) .

本研究旨在检测VEGF-C及其受体Flt-4（fms-liketyrosinekinase-4）在舌鳞癌中的表达，并探讨其临床意义。

PURPOSE : To study the expressions and clinical significance of urokinase-type plasminogen activator ( uPA ) and uPA receptor ( uPAR ) in tongue squamous cell carcinoma ( TSCC ) .

目的：研究尿激酶型纤溶酶原激活剂（uPA）及其特异受体（uPAR）在舌鳞癌（TSCC）中的表达及其与临床病理参数的关系。

Over expression of VEGF C could induce the dilatation and LVD increase of peri tumor lymphatic vessels , which maybe one of the mechanisms that VEGF C metastasis of TSCC through lymphatic vessels .

VEGFC能够诱导癌周淋巴管扩张，这可能是VEGFC增加舌鳞癌颈淋巴转移的机制之一。

NY-ESO-1 protein has higher expression in the well differentiated and the bigger tumor tissues of TSCC . It can be further explored as one of the basic research in multivalent vaccine immunotherapy of TSCC .

结论：1NY-ESO-1蛋白在中高分化及肿块较大的TSCC组织中表达较高，可以进一步探索其作为TSCC免疫治疗多价疫苗的基础研究依据之一。

We suggest that expression and distribution of Col ⅳ, FN and LN in TSCC reflect the injury degree of the basement membrane , correlate well with the differentiation , the invasive pattern and neck lymph node metastasis of TSCC .

提示：TSCC中ColⅣ、FN和LN的表达和分布反映了基底膜的损伤程度，与癌细胞分化程度、浸润方式及淋巴结转移密切相关。

In 19 cases of TSCC with p53 gene mutation , the mutation was located in exon 5 in 12 cases , in exon 6 in 7 , in exon 7 in 2 and in exon 8 in 15 , respectively .

突变的19例舌癌，12例出现第5外显子突变，7例出现第6外显子突变，2例出现第7外显子突变，15例出现第8外显子突变。