sah

Model group : No intervention was given after SAH .

模型组：蛛网膜下腔出血造模后不给予干预；

An Analysis of Specific SAH CT Manifestations in New Born

新生儿蛛网膜下腔出血特殊CT征象分析

Clinical analysis of primary SAH in 45 cases of senile patients

老年人原发性蛛网膜下腔出血45例临床分析

SAH management concluded this would first require a process of'benchmarking ' .

SAH管理总结，这个是基准评价的第一个需要的过程。

Analysis of SAH with Identified Cause in 80 Patients

明确病因的蛛网膜下腔出血80例分析

Negative result in CT can 't rule out SAH .

CT阴性不能排除SAH；

Comparative study of clinical manifestation , CT and MRA of SAH

原发性蛛网膜下腔出血临床、CT与MRA分析

Use of the lumbar puncture and CT in diagnosis of primary SAH

腰穿及头颅CT在诊断原发性蛛网膜下腔出血中的作用

CONCLUSION : GBE may relieve cerebral ischemic damage after SAH .

结论：GBE可减轻SAH后缺血性脑损伤。

Conclusion CTA is the first choice in early etiological diagnosis for SAH .

结论CTA可作为SAH早期病因诊断学的首选方法。

Image Enzymology Check of Newborn SAH High Risk Facter and Prognosis Analysis

新生儿蛛网膜下腔出血高危因素影像学检查及预后分析

This process resulted in performance measures that greatly enhanced SAH 's ability to improve productivity and quality .

这个过程导致极大的增强SAH改善生产力和品质能力的性能标准。

Operation in three weeks after SAH is the independently risk factor on vasospasm .

SAH后3周内手术是颅内动脉瘤术后脑血管痉挛发生的独立危险因素。

The relationship of different blood components to cerebral vasospasm and immune abnormalities after SAH

蛛网膜下腔出血后不同血液成分与脑血管痉挛及免疫炎症反应的关系

The thesis sum up the most new study advance about SAH induce CVS molecular mechanism .

文章总结了SAH诱发CVS分子机制的最新研究进展。

CT Diagnosis of Delayed SAH Post-trauma ( with 40 Cases Report )

外伤后迟发性蛛网膜下腔出血的CT诊断（附40例报告）

Methods Clinical data and lumbar puncture and CT of80 patients with primary SAH were analysed .

方法分析80例诊断为自发性蛛网膜下腔出血的临床表现、腰穿及CT检查。

Brain calcium content increased significantly from 1h to 24h after induction of SAH .

脑组织Ca2+含量在SAH后1h开始显著增加。

Aesthetic Management of Ruptured Cerebral Aneurysm with Hunt 's ⅳ~ⅴ Grade Sah

麻醉管理在Hunt'sⅣ~Ⅴ级破裂脑动脉瘤手术中的意义

Methods 118 patients with aneurysmal SAH were retrospectively reviewed with clinical and radiological data .

方法回顾本院近5年收治的118例动脉瘤性SAH病人临床及影像学资料；评估入院时临床因素，判定脑血管痉挛程度；

The cause of primary SAH was mainly rupture of saccular aneurysms .

其病因主要是颅内动脉瘤破裂；

Our results suggest that ET is an early important factor which induces CVS after SAH ;

提示：ET是SAH后继发脑血管痉挛（CVS）的早期重要因素；

Purpose : To raise the rate of CT diagnosis for delayed SAH post-trauma .

目的：提高对外伤后迟发性蛛网膜下腔出血的CT诊断率。

It can judge cause of SAH whether ruptured AN and site of AN .

由此可判断蛛网膜下腔出血及脑内血肿是否由颅内动脉瘤破裂所致，以及颅内动脉瘤的部位。

Brain endothelin-1 content increased statistically after SAH .

脑组织ET-1含量逐渐增加。

RESULTS Increased expression of PDGF BB was found in the arterial wall after SAH .

结果SAH后48h脑基底动脉明显痉挛，痉挛血管壁的PDGFBB表达增多；

The prognosis of this SAH was better .

预后良好。

38 aneurysms in 36 SAH cases were detected by CTA .

CTA发现动脉瘤36例38个。

Results newborn SAH was closed related with hypoxia , birth trauma and perinatal health care .

结果发现新生儿SAH与窒息缺氧、产伤、围生期保健意识等因素密切相关。

Objective To analyse the clinical features and cerebral angiography of subarachnoid hemorrhage ( SAH ) .

目的分析自发性蛛网膜下腔出血的临床与脑血管造影术的特点。