pkd

Different subtypes of PKD plays different role in various malignant tumor .

PKD的不同亚型在不同的恶性肿瘤中作用各异。

Experimental Study and Clinical Abservation of PKD for Inflammation of Skin

复方黄连皮康酊治疗皮肤炎症的实验和临床观察

Conclusions The main inheritance mode of familial PKD is autosomal dominant .

结论家族性PKD的主要遗传方式为常染色体显性遗传，在临床及遗传上可能存在异质性。

Present study proves that PKD is mainly regulated by the G protein coupled receptor ( GPCR ) .

目前研究证明，PKD在细胞内的激活主要受G蛋白偶联受体（GPCR）调控。

The PKD domain is mainly composed of P-sheet with little a-helix or other secondary structures .

PKD结构域则主要由β-折叠，几乎不含α-螺旋和其他二级结构。

Clinical Report on the Detection of 14 Cases of Polycystic Kidney Disease ( PKD ) in the Three Families Using B-Mode Sonography

B超发现三家族14例多囊肾临床报告

Objective To observe the effect of Pumpkin Distillable Subject ( PKD ) on antioxidation in mice induced by Pb .

目的观察染铅小鼠抗氧化系统的改变及南瓜提取物（PumpkinDistillableSubject，PKD）对抗氧化酶系统的影响。

Objective : To detect the differences in subcortical structures between patients with paroxysmal kinesigenic dyskinesia ( PKD ) and normal subjects during movement preparation and execution .

目的：比较发作性运动诱发性运动障碍（PKD）患者与正常被试者在运动准备和执行过程中大脑激活差异，尤其是皮层下结构。

Gene mutations in human gene can cause various diseases . The human autosomal dominant brachydactyly type B ( BDB ) and autosomal dominant polycystic kidney disease ( PKD ) are due to gene mutation on chromosome .

人类基因组上基因的突变可以引起各种各样疾病的发生，人的常染色体显性的B型短指和常染色体显性的多囊肾综合症就是由于染色体上的基因突变引起的。

Methods The clinical findings of the patients with PKD were collected and the pedigrees were investigated . Electroencephalography ( EEG ) or video-monitoring EEG and brain magnetic resonance imaging ( MRI ) or computerized tomographies ( CT ) were performed on the probands .

方法观察8个PKD家系的临床表现，进行家系调查分析，对患者进行脑电图或视频脑电图、头颅磁共振成像（MRI）或CT检查。