ich

Clinical analysis of 36 cases with acute ICH followed by gastrointestinal bleeding

急性自发性脑出血伴消化道出血36例临床分析

ICH model was induced by collagenase ⅶ in the model group and treatment group .

模型组与治疗组采用Ⅶ型胶原酶诱导脑出血模型；

This study shows that , the ICH is of cultural nature as well as economic nature .

本文研究认为，非物质文化遗产既具有文化性，又具有经济性。

The research indicated CAA has become the important pathological foundation of ICH .

研究表明CAA已成为ICH的重要病理学基础。

Recent studies have suggested that apoptosis involved in nerve cell death following ICH .

最近的研究表明，凋亡机制参与了脑出血后神经细胞的死亡。

The therapeutic object is to relieve secondary brain injury following ICH .

减轻出血后继发性脑损伤，营救神经元仍然是治疗的目标，而更好的理解脑出血的病理生理机制无疑是提高临床疗效的有效途径。

The study of the role of inflammation may find a new approach for the treatment of ICH .

对脑出血后炎症反应的作用进行研究，有望找到新的脑出血治疗的切入点。

To explore the initial factor of apoptosis may find a new therapeutic strategy for ICH .

探讨细胞凋亡的始动因素，可为脑出血寻找新的治疗策略。

The contribution of clinical factors to the temporal distribution of ICH was also evaluated .

同时评价临床因素对脑出血病人在时间分布上的影响。

An Improved Autologous Blood ICH Model of Rat : Double Injection and Double Withdrawal

一种改良大鼠自体血脑出血模型：二次注血/退针法

So It is significant to study on mechanism of iron toxic process and intervention in ICH .

故对于脑出血后铁的毒性作用及干预机制的研究意义重大。

Methods The ICH rat model was established by intracranial injection of ⅳ collagenase .

方法使用胶原酶法建立大鼠脑出血模型。

The competent culture department shall be responsible for conducting ICH investigations .

非物质文化遗产调查由文化主管部门负责进行。

The safeguarding of ICH is a new challenge that the whole international community faces .

非物质文化遗产的保护是整个国际社会必须面对的一个新的挑战。

Listing mechanism is an essential method in providing for the safeguarding of ICH .

名录制度是非物质文化遗产保护的基本方法。

Conclusion : For all the spontaneous ICH patients , DSA examination is necessary .

结论：自发性脑内血肿均应进行全脑DSA检查，阴性患者在血肿基本消失后行MRI扫描尽可能查清病因；

ICH models were induced by collagenase type ⅶ - S.

用Ⅶ-S型胶原酶诱导大鼠脑出血模型。

The mechanism of nerve cell death following intracerebral hemorrhage ( ICH ) is complex .

脑出血后神经细胞死亡的机制复杂。

Other mechanisms of neural injury must be invovled in ICH .

脑出血可能还存在其他的损伤机制。

Objective To approach to the best therapy for acute intracerebral hemorrhage ( ICH ) .

目的：探讨治疗急性脑出血（ICH）的最佳方法。

Hirudin could inhibit the formation of cerebral edema after ICH .

水蛭素可抑制ICH后的脑水肿形成，抗凝血酶治疗可能成为ICH后脑水肿的一种可行治疗方法。

Whether age affects brain injury after ICH has not been systematically studied in an animal model .

但老龄是否影响脑出血后脑损伤，目前尚未见有人在动物模型方面进行系统研究。

Among these , ICH of ethnic minorities accounted for a great proportion .

这其中，少数民族非物质文化遗产占了极大的比重。

The neuronal injury was obvious 2 days after ICH under transmission electron microscope .

电镜下观察到出血后2d神经细胞损伤明显。

Clinical Significance of S-100 Protein Dynamic Change for ICH Score of Acute Hemorrhagic Cerebrovascular Disease

S-100蛋白的动态变化对急性出血性脑血管病评价的临床意义

The relationship between activation of microglia and damage of nerve cell DNA after ICH

脑出血后小胶质细胞活化与神经元DNA损伤的关系

It is characteristic of high attack rate 、 disability rate 、 case fatality rate for ICH .

脑出血具有发病率高、病残率高、病死率高的特点。

Correlation between transcranial Doppler and cerebral perfusion pressure in acute localized ICH rabbit model

兔急性局限性颅高压模型经颅多谱勒频谱参数与脑灌注压相关性的研究

Modeling and administration Models of rats with ICH were made with collagenase-induced method .

造模及给药：运用胶原酶法建立大鼠脑出血模型。

The present study will observe the expression of IL-6 in the rat brain of ICH at different time point .

本研究拟通过对脑出血大鼠不同时间点脑组织表达IL-6的观察，探讨IL-6表达在大鼠脑出血后继发性脑损伤及脑水肿中的作用。