hypertrophied

Effect of mechanical stretch on connexin 43 gene in SHR hypertrophied ventricular myocyte

机械牵拉对自发性高血压大鼠肥大心室肌细胞连接蛋白43表达的影响

Kocher demonstrate the transplantation of stem cell can decrease apoptosis of hypertrophied myocytes in the peri-infarct region and improve cardiac function .

Kocher等报道移植造血干细胞可改善心肌梗死周边区肥大心肌细胞的凋亡。因此提出干细胞不仅具有分化作用，也可以通过一些途径抑制损伤部位细胞凋亡的发生。

Study on Ischemia - Reperfusion Injury in the Hypertrophied Myocardium

肥厚心肌缺血再灌注损伤与心脏肾素

These findings suggest that the protein synthetic activity was increased in the hypertrophied myocardial cells .

本实验动物的心肌细胞，正处在工作负荷增加，蛋白质合成旺盛的心肌细胞肥大代偿期。

Objective To search a simple , safe and effective osteotomy for hypertrophied mandibular angle .

目的探索一种简便，安全，有效的下颌角肥大截骨术。

Conclusion : ADM is activated as a circulation hormone in thy hypertrophied model .

结论：在此心肌肥厚模型中肾上腺髓质素作为循环激素被活化，可能是一种代偿性升高。

Conclusion Over-expressed endothelin-1 gene may contribute to ventricular remodeling in hypertrophied rats .

结论压力负荷性大鼠表达上调的ET1参与心室重构。

The Remodeling of Gap junction and Its Influences on Conduction Properties in Hypertrophied Left Ventricle

心肌肥大时间隙连接分布的改变及其对传导特性的影响

The Acute and Chronic Effects of Amiodarone on Currents in Isolated Ventricular Myocytes of Hypertrophied Rats

胺碘酮对大鼠肥厚心室肌细胞离子流的急性及慢性作用研究

Calcineurin A β mRNA expression and its role in hypertrophied right ventricle of rats caused by chronic hypoxia

钙调神经磷酸酶Aβ在缺氧性右心室心肌肥厚中的表达及作用

Results : It was found that the myocardial chymase like activity increased markedly in hypertrophied rat ( 2,6,12 weeks );

结果：术后2、6、12周心肌肥厚组大鼠心肌组织糜酶样活性较同期正常对照组显著增高（P＜0.01）；

Expression of Ang ⅱ receptors in excess load-pressured hypertrophied cardiac muscles of rats

压力负荷性大鼠肥厚心肌中AngⅡ受体的表达

[ Conclusion ] Atorvastatin can prevent partly gap junction remodeling of hypertrophied myocardium induced by pressure overload .

结论阿托伐他汀能部分防止压力负荷诱导的心肌肥厚时的Cx43重构。

To examine changes of collagen network and Matrix Metaloproteinases ( MMPs ) activity in hypertrophied rat myocardium .

探讨压力负荷增高性心肌肥厚时心肌胶原网络及心肌细胞外基质金属蛋白酶（MMPs）活性的变化。

The present study was to observe these two factors in the hypertrophied ventricles and aortas of sinoaortic-denervated rats .

本研究观察去窦弓神经大鼠肥厚心室和主动脉组织中这两因子的改变。

[ Objective ] To investigate the effect of atorvastatin on gap junction remodeling in hypertrophied myocardium induced by pressure-overload .

目的探讨阿托伐他汀对心肌肥厚时缝隙连接重构的影响。

Effect of atorvastatin on gap junction remodeling in hypertrophied myocardium Propranolol pretreatment protects ischemic hypertrophied myocardium in rat

普萘洛尔预处理对大鼠肥厚心肌缺血再灌注损伤的保护作用及其机制探讨阿托伐他汀对肥厚心肌缝隙连接重构的影响

Hypertrophied myocardium is prone to proarrhythmic toxicity and development of the torsade de points type of ventricular tachycardia .

肥厚心肌对于抗心律失常药物的致心律失常毒性非常敏感，容易发生尖端扭转型室速。

All 20 clones formed hypertrophied lenticels and adventitious roots by Day 6 to 14 of flooding .

20个无性系均在淹水6～14d后形成了膨大皮孔和不定根。

In hypertrophied myocardium and heart , KB-R7943 shows common pharmacologic profile but more intense than in normal myocardium and heart .

在肥厚心肌，KB-R7943表现出相同的药理作用，但抑制作用更强。

KB-R7943 completely blocked the activating effects induced by E-4031 in both of normal and hypertrophied cardiomyocytes .

R7943可完全阻断正常组和肥厚组心肌细胞由E-4031诱导的激动作用。尼卡地平不能阻断E-4031的增加作用。

In adult group , the slab , wedge and hypertrophied horn type was seen in 36 , 15 and 6 knees , respectively .

成年组板型36膝、楔形15膝、肥角型6膝。

Changes of characteristics of transient outward potassium current , inward rectifier potassium current and calcium current in single cells isolated from thyroxine induced hypertrophied rat hearts

甲状腺素诱发大鼠心肌肥厚时瞬时外向钾电流、内向整流钾电流和钙电流的变化

Objective To explore response of Na + / Ca 2 + exchange to β - adrenergic stimulation in hypertrophied myocytes and the possible mechanism .

目的探讨肥厚心肌细胞Na+/Ca2+交换对β-类肾上腺素能药物刺激的反应及发生这种改变的可能机制。

Results In impuberal group , slab type was seen in 26 , wedge type in 6 and hypertrophied horn type in 6 knees .

结果未成年组板型26膝、楔型4膝、肥角型6膝；

In contrast it is characterised with multi-channels and nonspecific disorder in the hypertrophied myocardium in the acquired heart disease .

而后天性心脏病的心肌肥大是多离子通道病变而非特殊性。

Inhibition of Na + / Ca + exchange induces the suppression in systolic and diastolic function of myocardium , especially in hypertrophied heart .

抑制心肌Na+/ca2+交换将导致心肌收缩和舒张功能降低，这种抑制作用在压力负荷型肥厚心肌表现更为显著。

Cell layers of powder chamber wall and middle-level were increased , tapetum cells were hypertrophied and microsporogenesis or microspore disaggregated ;

药室内壁、中层层数增加，绒毡层细胞肥大，造孢细胞或花粉母细胞分解解体；⑤由于花粉母细胞胼胝质壁不降解而无法释放出四分体小孢子；

Rapidly Activating Delayed Rectifier K + Current and Inward Rectifier K + Current in Cardiocytes from Hypertrophied Guinea Pig Hearts Induced by Thyroxine

甲状腺素诱导的豚鼠肥厚心肌中快速激活的延迟整流钾电流和内向整流钾电流离子通道特征

These results suggest that autophagy of cellular elements seems to play an important regulative role in hypertrophied process and dilated T-tubules actually reflect myocardial adaptive changes .

这些结果说明，细胞成份的自体吞噬降解在心肌肥大过程中起着重要调节作用，扩张的T管也反映了心肌细胞外吐加快的适应性改变。