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alad

  • 网络脱水酶;氨基酮戊酸脱水酶;氨基乙酰丙酸脱氢酶;氨基乙酰丙酸脱水酶;酮戊酸脱水酶
aladalad
  1. On relationship between blood lead , ALAD activity and FEP in different urban risk groups

    城市不同危险人群血铅与ALAD活性和FEP值的关系

  2. Lead sensitivity gene ALAD of newborns in electronic waste recycle area had various statuses .

    铅易感基因ALAD在电子垃圾拆解区新生儿中存在多态性。

  3. Study of ALAD and VDR gene polymorphisms associated with lead nephrotoxicity susceptibility

    δ-氨基-γ-酮戊酸脱水酶和维生素D受体基因多态性与铅肾毒性易感性的相关研究

  4. Study on Porphyrin Metablism , Blood Pressure and ALAD Genetic Polymorphism in Lead Exposed Workers

    铅暴露者卟啉代谢、血压和ALAD基因多态性的研究

  5. Bioinformatics analysis of banana ALAD gene .

    香蕉ALAD基因的生物信息学分析。

  6. This research so far had not discovered ALAD gene type correlation with newborns umbilical cord blood Lead levels . 4 .

    本研究未发现ALAD基因型与新生儿脐带血铅水平有相关性。

  7. In addition , the interaction of blood Pb with ALAD gene , which may affect Pb toxicity , was studied .

    2探讨环境因素与基因的交互作用对铅毒性效应的影响。

  8. The study on δ - aminolevulinic acid dehydratase ( ALAD ) polymorphism in occupational lead exposed workers

    铅接触工人δ-氨基酮戊酸脱氢酶基因多态性研究

  9. Conclusion The smeltery industry induced serious lead pollution problems , which caused elevated blood lead level and reduce δ - ALAD in the children .

    结论该铅冶炼厂已造成附近居民生活环境区域性铅污染,并对儿童造成铅危害。

  10. [ Objective ] To explore the association of polymorphisms of MGP gene and ALAD gene with lead poisoning in children .

    [目的]探讨基质γ-羧基谷氨酸蛋白(MGP)和氨基乙酰丙酸脱水酶(ALAD)基因多态性在儿童铅中毒发生过程中的作用。

  11. The gene polymorphism of δ - aminolevulinate dehydratase ( ALAD ) in 530 cases of Chinese Han population

    530例中国汉族人口δ-氨基乙酰丙酸脱水酶基因多态性分析

  12. Conclusion The frequency of the ALAD 2 allele in Chinese Han population was similar to that in Asian populations and lower than that in Caucasian population

    结论中国汉族人口的ALAD2等位基因频率与文献报道的亚洲人群的结果相近,而低于高加索人口

  13. An in vitro study of inhibitory activity of prothrombin to calcium oxalate crystallization The Association of Gene Polymorphisms of MGP and ALAD with Lead Blood Level in Children

    凝血酶原分子中γ-羧基谷氨酸对草酸钙结晶的抑制作用研究基质γ-羧基谷氨酸蛋白和氨基乙酰丙酸脱水酶基因多态性与儿童血铅的关系

  14. Objective To investigate the polymorphism of δ aminolevulinic acid dehydratase ( ALAD ) and the genetic susceptibility to lead toxicity in Uighur and Yi population in China .

    目的探讨我国维吾尔族、彝族人群δ氨基乙酰丙酸脱水酶(ALAD)基因多态性与铅遗传易感性的关系。

  15. [ Objective ] To explore the effect of δ - aminolevulinate acid dehydratase ( ALAD ) gene polymorphism on hormone levels of thyroid in workers occupationally exposed to lead .

    [目的]研究δ-氨基乙酰丙酸脱水酶(ALAD)基因多态性对职业铅接触工人甲状腺激素水平的影响。

  16. Conclusions It was suggested that the ALAD gene polymorphism modified the relationship between blood lead and ZPP and the VDR gene variants influenced the skull development in children living under lead polluted environment .

    结论在高水平铅暴露儿童中,ALAD多态性可影响铅的血液毒性效应,VDR基因的遗传变异改变铅对儿童颅骨发育的作用程度。

  17. Conclusion : Supplied Prophylactically or treat with Vitamin E ^ Vitamin C can low blood lead level , increase Fe level , recover activity of ALAD and Hb , decrease injury of blood system by lead .

    结论:在铅中毒的同时及其后,补充维生素C、维生素E可降低血铅,升高血铁浓度,恢复ALAD和Hb的活性,改善铅导致的血液系统损伤。

  18. Objective To explore the genetic polymorphism and the distribution of allelic frequencies of δ aminolevulinic acid dehydratase ( ALAD )( E.C.4.2.1.24 ) in Uighur and Yi population in China .

    目的探讨中国维吾尔族、彝族人群δ氨基γ酮戊酸脱水酶(ALAD)基因多态性和等位基因的分布规律。

  19. These genes are δ aminolevulinic acid dehydratase ( ALAD ) gene , vitamin D receptor ( VDR ) and hemochromatosis gene . Their relation to susceptibility to lead poisoning in humans was reviewed in this paper .

    δ氨基γ酮戊酸脱水酶基因、维生素D受体基因及血色素沉着病基因等遗传多态性可能造成儿童对铅毒性作用有不同的易感性,本文就此进行综述。

  20. Objectives To explore the relationship between δ - aminolevulinic acid dehydratase ( ALAD ) gene polymorphism and susceptibility of toxicity induced by lead ( Pb ) which attacks the major target organs such as nervous system , kidney and blood system .

    目的1探讨ALAD基因多态性与铅的神经毒性、肾毒性和血液毒性的易感性关系。

  21. After Na-DMS treatment the inhibited ALAD activity was markedly recovered to normal in most patients , but only a little in Ca-Na2-EDTA , since Ca-Na2-EDTA did not contain SH groups and induced much more zinc excretion in urine than Na-DMS .

    Na-DMS含有两个巯基,并且由尿排锌不多,治疗后,绝大多数患者的血ALAD活力恢复正常。