镉中毒

干预组小鼠淋巴细胞损伤结果均低于镉中毒组（P0.05）。

Intervention group mice lymphocytes injury results were lower than the cadmium poisoning group ( P0.05 ) .

目的：对新疆部分MT含量较高的地产植物MT进行配伍组合，观察高含量植物MT提取液对小鼠镉中毒的拮抗作用类型及其可能机制。

Objective : The part of plants which was produced in the location of Xinjiang with the high levels of MT was compatibility . we observed the type of the antagonism cadmium poisoning under the high concentration of plant extract on mice MT and its possible mechanism .

大鼠DNA氧化损伤及抗氧化酶活力变化与慢性镉中毒

DNA oxidizing damage and activity of antioxidative enzymes in rats with chronic cadmium poisoning

结论：镉中毒可导致大鼠脂质过氧化和DNA氧化损伤。

CONCLUSION : Cadmium poisoning can result in lipid peroxidation and DNA oxidative damage in rats .

镉中毒肝脏过氧化氢（H2O2）定位及抗氧化系统的变化

Study on Hydrogen Preoxide ( H_2O_2 ) Localization and Antioxidative System Changes of Hepatocytes After Cadmium exposure

用200尾（108±12）g的草鱼作实验性镉中毒实验。

200 Grass carps with ( 10.8 g ± 1.2 ) g were made experimental cadmium ( Cd ) poisoning .

目的观察甘草甜素（GL）和齐墩果酸（OA）对大鼠亚慢性镉中毒性肾损害组织病理形态的影响。

Objective To observe effects of glycyrrhizin and oleanolic acid on renal histopathological changes induced by subchronic cadmium exposure in rats .

目的：研究急性镉中毒大鼠肝细胞中金属硫蛋白（MT）在超微结构水平的分布。

Objective : To investigate the ultrastructural intracellular distribution of metallothionein ( MT ) in acute cadmium exposed rat liver , especially in liver parenchymal cells .

结果表明DTPA、EDTA和8102对氯化镉中毒的小鼠有明显解毒作用，尤以DTPA、EDTA解毒作用为佳。

The results indicated that DTPA , EDTA and 8102 had obvious detoxication effect after cadmium chloride intoxication , especially DTPA and EDTA .

结论氧化损伤是镉中毒机制的一部分，LA和Tau对镉致肾氧化损伤的恢复具有一定的促进作用。

Conclusions Oxidative damage is a part of the mechanism of cadmium toxicity . LA and Tau show some promoting effect on healing after sub-chronic damages induced by Cd .

背景与目的：观察急性镉中毒大鼠肾皮质中血红素氧化酶-1（HemeOxygenase-1，HO-1）表达的变化。

BACKGROUND & AIM : To observe the changes of heme oxygenase-1 ( HO-1 ) expressed in renal cortex of rats with acute cadmium exposure .

用3日龄雏鸡150只，作急性和亚急性镉中毒病理学观察，急性毒性的LD50为8448mg/kg体重。

Three day old broiler chickens were made experimental Cd poisoning . LD50 of acute Cd poisoning was 84 48 mg / kg body weight .

慢性镉中毒对小白鼠肾脏的损害

Chronic Cadmium Intoxication Induced Injury on the Kidney of the Mice

镉中毒时肾细胞内微量元素含量的改变

Changes in Trace Elements Contents of Renal Cells in Cadmium Poisoning

家蝇幼虫消化道的解剖和镉中毒时的症状观察

Dissection of alimentary canal of housefly larvae and its changes in

急性镉中毒的肝脏损伤机制及金属硫蛋白的保护作用

Mechanism of liver injury in cadmium toxication and protection of metallothionein

慢性镉中毒对小白鼠肝脏损害的观察

Chronic Cadmium Toxication Induced Injury on the Liver of the Mice

镉中毒慢性肾衰小鼠模型的建立与评价

Establishment and evaluation of the cadmium-induced mouse model of chronic renal failure

急性镉中毒大鼠肝脏金属硫蛋白的免疫电镜定位

Ultrastructural localization of metallothionein in acute cadmium exposed rat liver

大鼠附睾组织镉中毒及防护机制的研究进展

Progress in Study of Cadmium Toxicity and Protective Mechanism in Rat Epididymis

目的探讨镉中毒性肾损害的细胞分子学机制。

Objective To study the cell molecular mechanism of cadmium-induced renal damage .

慢性镉中毒肾小管上皮细胞凋亡形态学研究

Morphological Study on Apoptotic Cells of Renal Proximal Tubules Exposed Cadmium Chronically

但到目前为止，慢性镉中毒尚无有效的治疗方法。

But so far , no effective therapy for chronic cadmium poisoning .

鸡实验性镉中毒病的研究

The Investigation on Hens ' Toxic Disease of Cadmium Chloride

实验性镉中毒大鼠尿钙症及其机理的探讨

Study of Calciuria and Mechanism in Experimental Cadmium Poisoned Rat

慢性镉中毒小鼠肾脏的超微结构观察

The ultrastructural observations in the kidneys of chronic cadmium toxication

镉中毒致肺损伤的病理形态学观察

Pathologic survey on lung impairment induced by cadmium poisoning

尿液低分子量蛋白测定在慢性职业性镉中毒诊断中的意义及方法评定

Significance of Urinary Low Molecular Protein Determination in Diagnosing Chronic Occupational Cadmium Poisoning

急性氯化镉中毒肾损害大鼠尿γ－谷氨酰转换酶及其同工酶

Rat urinary γ - glutamyltransferase and its isoenzyme in cadmium chloride-induced acute nephropathy

慢性镉中毒小鼠胰腺光镜下结构的观察

A Study on Histological Structure of Pancreas of Mice Chronically Intoxication with Cadmium