线粒体内膜

解耦联蛋白3（UCP3）是位于线粒体内膜的质子转运体，其在适应性产热、体重调节和能量代谢中发挥重要作用。

Uncoupling proteins are proton transporters in mitochondrial inner membrane . It plays an important role in thermogenesis , body weight regulation and energy metabolism .

能量化时线粒体内膜表面电荷的变化

Change in surface charge of mitochondrial inner membrane upon energization

线粒体内膜蛋白ANT在幽门螺杆菌致胃癌细胞凋亡中的表达

The effect of Helicobacter pylori on expression of ANT mRNA in gastric carcinoma cells

测定心肌细胞线粒体内膜的荧光偏振度及血清丙二醛（MDA）含量。

The fluorescence polarization of myocardial mitochondrial membrane and the content of serum MDA were detected .

力竭运动状态下线粒体内膜流动性及呼吸链酶活性发生变化，使氧化磷酸化过程受阻，致使ATP生成减少，影响了运动能力。

The inner mitochondria membrane ( IMM ) fluidity and activity of the respiration chain changed during exhaustive exercise .

发现心肌和骨骼肌线粒体内膜荧光偏振值和微粘度均较安静时增高，示内膜流动性显著降低（均P＜0.01）；

The results showed that the fluorescence polarization and microviscosity values of IMM in rat heart and skeletal muscle were higher than the resting values ( p < 0.01 ), which meant the IMM fluidity decreased obviously .

腺嘌呤核苷酸载体（ANT）是一种位于线粒体内膜上的转运蛋白，它在组织细胞能量产生和消耗的耦联中起关键性作用。

Adenine nucleotide translocator is a kind of transport protein in the inner mitochondrial membrane , it plays a key role in the coupling of energy production and consumption .

用ANS荧光强度的变化反映大鼠心肌线粒体内膜的能量偶联功能。

The function of energy coupling was determined by the changes of fluorescence intensity of ANS .

文中报告了焦痂毒性提取物D1对大鼠肝线粒体内膜膜电位的影响。

The effects of the toxic extract , D1 of burn eschars on the mitochondrial transmembrane potential were observed in rats .

Bcl-2的表达产物位于线粒体内膜、核膜及内质网膜，它可通过对脂质过氧化的抑制作用而发挥其抑制细胞凋亡的功能。

Bel - 2 expressing product is localized at mitochondrion endometrium , nuclear membrane and endoplasmic reticulum membrane , it can inhibit lipid peroxide , so inhibit apoptosis and prolong the cell life .

解偶联蛋白3是一种线粒体内膜上的转运蛋白，可以使氧化过程与ADP磷酸化过程解偶联。

Uncoupling protein 3 ( UCP3 ) was mitochondrial membrane transporters involved in themogenesis , which uncoupled the oxidation and phosphorylation of ADP on respiration chain .

PBC特异的AMA主要针对位于线粒体内膜的三个酮酸脱氢酶复合物家族（OADC）中的各个亚单位，即线粒体M2抗原的主要成分，其中最主要针对丙酮酸脱氢酶复合物E2亚单位（PDC-E2）。

AMA in PBC are directed to the three ketoacid dehydrogenase complexes , and predominantly to E2 subunit of pyruvate dehydrogenase complex ( PDC-E2 ) .

目的：解偶联蛋白2（UncouplingProtein2，UCP2）是线粒体内膜解偶联蛋白质家族成员。

Objectives : Uncoupling protein 2 ( UCP2 ) is a member of the anion carrier family localized in the inner mitochondrial membrane .

研究了鼠肝线粒体内膜体呼吸链复合体Ⅱ＋Ⅲ的H~＋／2e比与Δψ的相关性及其调节因素。

A study on the relationship between H ￣ + / 2e ratio and Δψ across inner mitochondrial membrane in rat liver mitochondria was carried out .

结论：冷适应不仅能使棕色脂肪线粒体内膜UCP含量增加，而且能使其活性增加。

CONCLUSION : Cold acclimation not only increase concentration of UCP in mitochondria inner membrane of rat brown fat , but also increase its activity .

UCPs借助于其信号序列，通过Tim10/Tim12/Tim22通道向线粒体内膜定向转运。

UCPs are directionally transported on mitochondrial endo-membrane via Tim10 / Tim12 / Tim22 channel by their signal sequences .

目的：探讨决明子、黄芪对大鼠棕色脂肪线粒体内膜解偶联蛋白（UCP）活性与含量的影响。

Objective : To evaluate the effects of Cassia seed and Astragalus on the content and activity of uncoupling protein ( UCP ) in mitochondria intima of brown fat in rats .

近来研究表明：解偶联蛋白2（UCP2）广泛存在于组织细胞的线粒体内膜，具有抑制活性氧产生的功能。

Recently , it has been reported that Uncoupling Protein 2 ( UCP_2 ) is widespread in mitochondrial inner membrane of tissue including cadiocyte , and can decrease the production of ROS .

结论：黄单胞菌L4毒素破坏线粒体内膜结构，从而使ATP代谢不能正常进行，叶面气孔关闭，而且蒸腾作用的热量无法散发，使叶片坏死萎蔫。

Heat of transpiration cannot be emitted and induce leaf to wilt . Conclusion : Xanthomonas L4 phytotoxin can disrupt mitochondrial to inhibit oxidation and phosphorylation to couple with each other , so ATP can not be formed normally result in leaves to death .

线粒体内膜上F1-ATP合酶只有十几个纳米，但能量转化效率却高得惊人，几乎达到100%！

F1 - ATPase has a diameter of about 10 nm , but very high efficiency of energy transformation , nearly 100 % !

本实验旨在探讨急性缺血再灌注后心肌细胞线粒体内膜解偶联蛋白2表达的变化。结论：缺血前给予EIPA对在体心脏缺血再灌注损伤具有保护作用，与缺血预处理具有类似效果。

The present experiment is to investigate the expression of UCP_2 in acute myocardial ischemic-reperfusion injured rat heart . Conclusion : Administration of EIPA before ischemia and ischemic preconditioning have protective effect on myocardial ischemic-reperfusion injury .

失血性休克致鼠心肌线粒体内膜的损伤

Hemorrhagic Shock-induced Damage in the Inner Membrane of Rat Myocardial Mitochondria

脂质膜体与线粒体内膜的融合

Fusion between Liposome and the Inner Membrane of Mitochondria

呼吸链底物和抑制剂对线粒体内膜流动性的影响

Effects of substrates and inhibitors for respiratory chain on membrane fluidity of mitochondrial inner membrane

琥珀酸脱氢酶与线粒体内膜的结合&泛醌的影响

The binding of succinate dehydrogenase with the mitochondrial inner membrane & the effect of ubiquinone

上述结果表明：线粒体内膜流动性与其功能密切相关。

These results indicated that membrane fluidity and function of the mitoplast were closely related .

线粒体内膜能量相关转运子在大鼠压力负荷模型早期心肌重塑中的表达

The expressions of energy-related translocators within mitochondrial inner membrane during early myocardial remodeling under pressure load in rats

冷适应对大鼠棕色脂肪组织线粒体内膜解偶联蛋白与嘌呤核苷三磷酸结合的影响

Effect of cold acclimation on GTP binding to uncoupling protein in mitochondria inner membrane of rat brown fat

冷暴露大鼠心肌线粒体内膜流动性及寡霉素敏感蛋白变化

Changes of Intimal Fluidity and Function of the Oligomycin Sensitive Protein of Myocardial Mitochondria in Rats During Cold Exposure

冷适应2,3周时线粒体内膜蛋白质电泳图谱呈现明显变化，冷适应组线粒体内膜有新的蛋白质条带出现。

The electrophoresis pattern of the inner membrane proteins in mitochondria from cold adapted rat livers revealed some new bands .