neuroglial cell

If want to changed the stat , must be operatived decrease secondary injury , limited proliferation rate of neuroglial cell , promoted neuron regeneration , after spinal cord injury .

只有有效的降低脊髓损伤后的继发性损害、限制神经胶质细胞的增生速度、促经神经元的再生、才能防止脊髓坏死空洞和胶质瘢痕的形成。

Compared with that of respective control groups , the released content of NO from macrophages , neuroglial cell of the NGF groups was decreased in the normal mice and the NOD mice .

与各自对照组相比，正常小鼠、NOD小鼠各实验组巨噬细胞、神经胶质细胞NO释放量均减小。

Research progress in the mechanisms of regulating synaptic plasticity for neuroglial cell

神经胶质细胞调节突触可塑性机制的研究进展

BFGF could be expressed in neuron and in neuroglial cell , specially in cell plasm and membran .

bFGF在神经细胞和神经胶质细胞内均可表达，以细胞浆及胞膜表达阳性染色为主。

Study on the Plasticit Change of Neuroglial Cell and the Expressing Rule of bFGF During Convalescence of Focal Cerebral Ischemia Penumbra

局灶脑缺血恢复期半影区小胶质细胞可塑性变化和bFGF表达规律的研究

In addition , close approximations or direct contacts between NT-LI terminals and blood vessels or neuroglial cell bodies were observed . bearing oil interface

此外，神经降压肽样免疫反应轴突终末可与血管和神经胶质胞体相邻或者直接接触。轴承与润滑油的接触面

The ultrastructure evidence for PFn-striatal axonal terminals in rats In addition , close approximations or direct contacts between NT-LI terminals and blood vessels or neuroglial cell bodies were observed .

丘脑束旁核-纹状体神经元轴突终末的超微结构证实此外，神经降压肽样免疫反应轴突终末可与血管和神经胶质胞体相邻或者直接接触。

The processes of neuroglial cell were increased and a special lamellated membranous , myelin sheath-like structures enclosing dendrites , axon terminals and even synapses were found in old rats .

神经胶质细胞突起增多，并可进一步形成包绕树突、轴突终末、甚至突触的多层膜环绕的髓鞘样结构。

Conditions such as brain injury , inflammation and infection could cause extensive immunocyte to enter brain or activate neuroglial cell and neuron to excrete proinflammatory cytokines that took part in formation and developmentof brain e-dema as well as damage plerosis and regeneration of neuron .

脑损伤及炎症、感染等情况可导致外周免疫细胞进入脑内，或激活神经胶质细胞、神经元等分泌炎性细胞因子，参与脑水肿的发生、发展以及神经元损伤、修复与再生。

S100 β is one kind of the calcium binding proteins . As growth factor of neuraxon , it is excreted by neuroglial cell , and distributing in nerve tissue extensively . Although S100 β has very important values neurophysiological , it also has neurotoxicity with excreting overmuch .

S100β是一类钙离子结合蛋白，是轴突的生长因子，由神经系统胶质细胞分泌，广泛分布于神经组织中，在正常情况下发挥重要的生理作用，但分泌过高具有神经毒性。