ip3

45 rabbits in the analysis of contents of IP3 and camp , and all had 10 drop out .

三磷酸肌醇及环磷酸腺苷含量分析数均为45只，均脱失10只。

Methods : IP3 and calcium concentration were determined by the use of binding assay and from the change of Fura 2AM fluorescence , respectively .

方法：使用结合法测定三磷酸肌醇，根据Fura－2AM荧光变化测定钙离子浓度。

The Design and Simulation of High IP3 Microwave LNA

高IP3微波低噪声放大器的仿真设计

METHODS : [ 3H ] IP3 competitive protein binding assay .

方法：[~3H]IP3竞争性蛋白结合实验。

Method of Measuring High IP3

高IP3的测试方法

The metabolite of IP3 had no obvious effect on BK channels .

IP3的降解产物对通道没有明显的激活作用。

Conclusions 5-HT acts on guinea pig colon myocytes through IP3 pathway .

结论5-HT主要通过IP3途径作用于豚鼠结肠平滑肌细胞：①可增加钙电流和使细胞去极化引起平滑肌细胞收缩。

The activation of IP3 on BK channels did not decline .

IP3对通道的激活作用不随时间而衰减；

In this paper , the relationship between the dual tone dynamic range and IP3 is introduced , and then a design method is described .

从双音动态范围与IP3的关系出发，介绍了大动态范围接收机的设计方法。

P1 dB and IP3 are key parameters for the design of low noise amplifier . So correct analysis and optimization of them is very indispensable .

对低噪声功率放大器的设计，1dB压缩点（P1dB）和3阶截取点（IP3）是其2个关键参数，对他们进行正确的分析和优化是非常必要的。

These data suggest that IP3 plays an important role in Ca ( 2 + ) - overloading forming of acute renal ischemic reperfusion injury .

提示，细胞内IP3对急性肾缺血及再灌流损伤中肾组织细胞内钙超负荷的形成具有重要作用。

IP3 pathway activation can increase intracellular calcium and fat accumulation ; on the other hand , IP3 pathway suppression reduce fat accumulation .

说明IP3通路激活可引起胞内钙离子升高，脂肪蓄积增加；反之，脂肪蓄积减少。通过IP3通路，膳食钙可抑制胞内钙离子增加。

( Ca2 + ) I was determined by Fura-2 fluorescin load technic , IP3 was determined by competitive protein binding method .

Fura-2荧光负荷技术测（Ca2+）i，竞争性蛋白结合法测定IP3浓度。

IP3 pathway involved in the early release of Ca2 + from ER induced by A β 25-35 , the activation of PLC may involve in the process .

Aβ25-35可通过IP3途径产生IP3作用于IP3R而引起内质网钙的释放，磷脂酶C的活化可能参与了上述过程。

Shuai and P.Jung Ca2 + oscillation model , the dynamical IP3-Ca2 + oscillation model connected with varying IP3 concentration was obtained .

Shuai和P.Jung钙振荡模型，得到与IP3浓度相关的动态IP3-Ca2+振荡模型。

It is a complex signal network surrounding the important second messenger molecular IP3 and Ca2 + - related signal in plant germination , growth , procreation , senescence , and responses .

它是围绕重要的第二信使分子IP3并与钙信号密切相关的一个信号网络，既能够影响基因表达，又参与细胞对外界环境变化的响应，还参与激素作用。

AIM : To detect inositol 1,4 , 5-triphosphate ( IP3 ) formation of pregnant and nonpregnant human myometrial cells induced by acetylcholine ( ACh ) .

目的：检测乙酰胆碱（acetylcholine，ACh）对人妊娠与未妊娠子宫颈平滑肌细胞生物合成肌醇1,4,5三磷酸（inositol1,4,5-triphosphate，IP3）的影响。

Objective To synthesize and identify structure of the active peptide ( IP3 ) with high affinity for MDM2 , providing this structural information for designing anti-cancer non-peptide molecule based on the MDM2-p53 complex .

目的以MDM2p53复合物为靶标，固相合成与MDM2有高亲和力的活性多肽（IP3），并测定其二级结构，为设计具有抗癌活性的非肽小分子提供结构信息。

Conclusion 1,25 ( OH ) - 2D-3 could stimulate the intracellular calcium release channel of inositol triphosphate ( IP3 ) receptor open in MCC in vitro and increases the level of intracellular calcium concentration .

结论1,25（OH）2D3能刺激髁突软骨细胞内三磷酸肌醇受体（IP3R）钙释放通道开放，使细胞内钙离子水平显著升高；

Intracellular 1 , 4 , 5-trisphosphate ( IP3 ) and cyclic adenosine monophosphate ( cAMP ) were extracted , their concentrations were measured by liquid scintillation technique and gamma scintillation counter respectively .

先分别提取细胞内三磷酸肌醇（IP3）及环磷酸腺苷（cAMP），分别应用液体闪烁测量法、γ&闪烁记数仪测定其含量；

Conclusion The decrease of neurotrophin factor 3 in rats after chronic stress can promote the death of nerve cell , meanwhile neurotrophic factor 3 can regulate the death of nerve cell through IP3 / Akt and Pas / MAPK .

结论慢性应激后大鼠神经营养因子3的下调促进了神经元细胞凋亡。同时认为神经营养因子可能是通过磷酸肌醇3-激酶/Akt和Ras/MAPK途径调节神经元的凋亡。

Conclusions These results indicate that the effect of selenium improving insulin sensitivity may be related to phosphatidylinositol PI3K signalling pathway . The effect of regulation of IP3 by selenium is not as effective as that by insulin , which may explain the difference of effect between selenium and insulin .

结论亚硒酸钠改善青春期胰岛素抵抗大鼠的胰岛素敏感性，可能与改善肌醇磷脂PI3K信号通路有关，而对IP3水平的调节作用不及胰岛素。