hyperammonemia

Conclusion Proteus infection may be one of the inducing factors to hyperammonemia in cirrhotic patients .

结论变形杆菌感染是肝硬化患者高氨血症发生的影响因素之一。

Prevention of secondary complications : medical attention during intercurrent infections to prevent hyperammonemia .

对并发症的预防：在间发性的感染中应有医疗注意防止高血氨。

Methods 137 decompensated cirrhosis patients with hyperammonemia were investigated . Blood ammonia level was detected in all patients .

方法对137例合并高氨血症的肝硬化失代偿患者的Hp感染情况及血氨浓度进行观察，并探讨二者与肝硬化临床表现之间的关系。

The drug is also approved for use as maintenance therapy for chronic hyperammonemia that results from NAGS deficiency .

同时还批准该药物作为NAGS缺乏所致慢性高氨血症的维持治疗药物。

The Relationship of Aquaporin-4 mRNA and Protein Expression with Brain Edema in Liver Cirrhosis Rat Models with Hyperammonemia

氨负荷下肝硬化大鼠模型水通道蛋白-4mRNA及蛋白的表达与脑水肿关系的研究

Objective : To make the diagnosis and treatment of hyperammonemia due to ornithine transcarbamylase ( OTC ) deficiency .

目的：探讨OTC缺陷的临床、生化与遗传特点及诊断治疗方法。

Use of other ammonia-lowering therapies with Carbaglu during episodes of acute hyperammonemia is recommended .

建议在急性高氨血症发作期间与其他降低血氨的治疗联合应用。

Objective To investigate the incidences of urea cycle defects ( UCDs ) in the patients with hyperammonemia and study their etiology , clinical and laboratory features .

目的探讨尿素循环障碍的发生情况、病因、发病、临床经过与实验室特点。

Objective To study the curative effect of the colon perfusion dialysis for the decompensated hepatocirrhosis with intractable ascites , endotoxemia , hyperammonemia and hyponatremia .

目的研究结肠灌注透析对失代偿性肝硬化顽固性腹水及内毒素血症、高氨血症、低钠血症的治疗效果。

The episodes of hyperammonemia are similar to those seen in the acute neonatal form , but the initial neurologic findings may be more subtle because of the older age of the affected individuals .

血氨过多的发作和急性新生儿型相似，但是最初的神经科症状可能比较轻微因为患者发病时年龄较大。

Conclusion : 1 . Increase of the blood ammonia was the main cause for the brain energy metabolic abnormality and AQP-4 mRNA and protein expression , hyperammonemia was the key occurrence and development of the hepatic brain edema .

结论1.在肝功能衰竭中血氨增高是导致脑内能量代谢异常、星形胶质细胞内AQP-4mRNA和蛋白表达增加的主要因素，高氨血症是肝性脑水肿发生和进展的核心环节；

Results : All of 4 patients had serious malnutrition during liver transplantation caused by disorders such as blood sugar fluctuation , electrolyte disturbance , hypokalemia , hyperammonemia , mental disorder , hyperglycemia , infusion and hypoproteinemia .

结果：4例接受肝移植病人术前存在不同程度营养不良，术中易发生血糖波动、电解质紊乱，术后易出现高氨血症、低钾血症、精神障碍、血糖升高、液体超载、低蛋白血症等。