galactosamine

Indomethacin induced hepatic protection against galactosamine toxicity in rats

消炎痛诱导大鼠肝脏对半乳糖胺损伤的保护作用

Methods : 30 Wistar rats were injected D galactosamine to induce AHF model .

方法应用D-半乳糖胺腹腔注射诱导Wistar大鼠呈急性肝脏衰竭。

Arachidonic Acid Metabolism in Galactosamine / Endotoxin Induced Acute Liver Injury in Rats

急性氨基半乳糖／内毒素肝损伤鼠的花生四烯酸代谢

Methods ALF in mice was induced by a single intraperitoneal injection of D galactosamine .

方法D氨基半乳糖诱导小鼠急性肝衰；

Method : The D galactosamine ( D Gal ) was used for rats and mice experimental hepatic injury .

方法：采用D－氨基半乳糖（D－Gal）致大鼠小鼠实验性肝损伤。

A liver cirrhosis model of rat was established by galactosamine ( Galn ) induction .

采用半乳糖胺（Galn）诱导大鼠肝硬化。

The Effect of cHSS on the Surviving Percentage of Hepatic Necrosis Rats Treated with D & galactosamine

新生牛肝再生刺激因子对大鼠药物性急性肝功能衰竭存活率的影响

Therapeutic mechanism of hepatic stimulator substance for experimen - tal acute liver failure induced by D - galactosamine

肝再生刺激物质救治D-氨基半乳糖肝衰大鼠的机理

The Relationship between Galactosamine Hepatotoxicity and Cortical hormone

半乳糖胺盐致肝损害与肾上腺皮质激素水平的关系

METHODS Acute hepatic injury model induced by tetrachloromethane and galactosamine was studied in this paper . Carbon particle clearance and serum haemolysin was determined .

方法：采用四氯化碳、D-氨基半乳糖化学性肝损伤模型及碳粒廓清和血清溶血素等免疫学模型的动物实验。

The level of serum glucosamine and galactosamine were determined in 19 female patients with systemic lupus erythematosus by the second derivative spectrophotometry method .

采用二阶导数分光光度法，对19例女性SLE患者血清氨基葡萄糖（GlucosamineGlus.）和氨基半乳糖（GalactosamineGals.）进行检测。

Method : Mouse acute liver injury model was induced by D galactosamine ( D GL ) or carbon tetrachloride ( CCl 4 ) .

方法：分别采用D半乳糖胺（DGL）和四氯化碳（CCl4）诱导小鼠发生实验性急性肝损伤。

Methods : 40 rats were randomly divided into a normal group ( 10 rats ) and acute liver injury models ( 30 rats ) indued by G galactosamine ( D GalN ) .

方法：用D氨基半乳糖（DGalN）制备大鼠急性肝损伤模型。

Mothods : The biochemical indexes and histopathological charges of liver injury in mice caused by toxicant chemicals [ D galactosamine ( D Galn ) and CCl 4 ] were determined .

方法：采用化学毒物D半乳糖胺和四氯化碳致小鼠肝脏损伤，测定生化指标及观察病理组织学改变。

Methods The model of experimental liver injury was produced by using carbon tetrachloride ( CCl 4 ) and D galactosamine ( D Gal ) in mice . The content of serum GPT and LDH were measured respectively .

方法实验采用四氯化碳（CCl4）和D氨基半乳糖（Dgal），造成动物急性实验性肝损伤，测定小鼠和大鼠血清GPT和LDH，并进行组织形态学观察。

It also agglutinated the peripheral lymphocytes cells and S 180 tumor cells from mouse . The hemagglutination on rabbit erythrocytes was inhibited by lactose , raffinose , galactose ,α methyl galactose ,β methyl galactose and N acetyl galactosamine .

它还可以凝集小鼠脾脏淋巴细胞和小鼠S180肉瘤细胞，对兔红细胞的凝集作用可被乳糖、棉子糖、半乳糖、α甲基半乳糖、β甲基半乳糖和N乙酰半乳糖胺所抑制。

Objective To study the role of tumor necrosis factor α( TNF α) and caspase 3 expression on hepatocyte apoptosis in experimental model of fulminant hepatic failure ( FHF ) Methods Mouse experimental model of FHF was induced by lipopolysaccharide ( LPS ) and D galactosamine ( D GalN ) .

目的研究肿瘤坏死因子α（TNFα）与caspase3表达在实验性暴发性肝衰竭（FHF）中对肝细胞凋亡的作用。方法用脂多糖和D氨基半乳糖制备FHF小鼠模型；