Syncytiotrophoblast

Conclusion : HCV persistent infection suppressed the biological behavior of human placental syncytiotrophoblast in vitro .

结论：HCV持续感染可抑制体外培养人胎盘合体滋养层细胞包括侵袭力和激素合成、分泌功能在内的多种生物学功能。

Western blot was applied to determinate the expression of IDO protein on cultured syncytiotrophoblast ;

westernblot检测体外培养的合体滋养层细胞IDO蛋白质表达；

The Analysis of Fatty Acid Composition in Syncytiotrophoblast Plasma Membranes of Human Placenta

人体胎盘合体滋养细胞质膜的脂肪酸组成分析

Result Immunolocalization of VEGF in all cases was observed mainly in syncytiotrophoblast .

结果VEGF主要表达于胎盘绒毛滋养细胞；

Relationship Between Matrix Metalloproteinase-9 Expression in Syncytiotrophoblast and Placenta Pathological Change of Intrauterine Growth Restriction

胎儿生长受限胎盘病理改变与绒毛滋养细胞基质金属蛋白酶9的表达关系

Study on the Relation between Syncytiotrophoblast Microparticles and Pathogenesis of Preeclampsia

胎盘合体滋养细胞膜微粒与子痫前期发病机制的关系研究

Results : KiSS-1 was mostly found in syncytiotrophoblast .

结果KiSS-1主要位于胎盘绒毛小叶的合体滋养细胞。

Objective To study the relationship between matrix metalloproteinase-9 in syncytiotrophoblast and placenta pathological change of fetal growth restriction .

目的了解胎儿生长受限（fetalgrowthrestriction，FGR）者胎盘病理改变与胎盘组织中基质金属蛋白酶9（MMP-9）表达水平变化的关系。

This leads to oxidative stress and damage to the placenta , specifically to the syncytiotrophoblast .

这会导致胎盘的氧化应急和损伤，特别是对于合胞体滋养层。

Villi syncytiotrophoblast layer and cytotrophoblast and loose in the middle of stromal part .

绒毛组织由合体滋养层及细胞滋养层及中间的疏松间质三部分所构成。

The results show : in hydatidiform moles , syncytiotrophoblast of floating villi , LN is negative ;

结果显示：LN在葡萄胎中的游离绒毛合体滋养细胞（LNR）呈阳性着色、LN呈阴性；

Future studies will determine whether carbon monoxide can prevent syncytiotrophoblast death in animal models and whether other approaches similar to carbon monoxide may provide feasible protection .

进一步的研究需要确定一氧化碳是否可以在动物模型中抑制合胞体滋养层的死亡，以及是否有和一氧化碳相似的途径存在保护作用。

Leptin expressed on the placenta villous syncytiotrophoblast .

Leptin主要表达于胎盘合体滋养层细胞的胞浆中。

Mainly in epithelial cells of the amnion , chorionic trophoblast nucleus , syncytiotrophoblast membrane and cytoplasm of placenta .

结果AQP8蛋白在羊水过少组和正常羊水组产妇的胎盘、胎膜组织中均有表达，主要分布于羊膜上皮细胞、绒毛膜滋养细胞核胎盘合体滋养细胞的细胞膜和细胞质中。

Conclusion One of the possible mechanism involved in perinatal of ICP is apoptosis caused by overexpression of bax in syncytiotrophoblast leading placental dysfunction .

结论ICP患者胎盘合体滋养细胞bax基因过度表达，胎盘细胞凋亡率升高，影响胎盘功能，可能是ICP并发围生儿缺氧的病理生理基础之一。

It requires enough of hormone secretion , syncytiotrophoblast formation , and the synchronal development of blastocyst and uterine endomembrane and also .

胚胎植入的成功必须具备许多条件，需要足够的激素分泌，合体滋养层细胞形成，以及胚泡与子宫内膜同步发育与相互配合等。

When syncytiotrophoblast cells die , they are released into the maternal circulation , initiating a cascade of inflammation that can damage maternal organs .

当合胞体滋养层细胞死亡，他们就会释放到母体的血液循环中，从而引起炎症级联反应，进而损害母体的器官。

Results HB-EGF expression was localized in the cytoplasm of cytotrophoblast and syncytiotrophoblast , but no expression was present in those with spontaneous abortion .

结果HBEGF主要存在于细胞质，合体滋养层和细胞滋养层均有表达，部分流产滋养细胞不表达。

CONCLUSION : HCV persistent infection suppressed the synthesis and secretion of MMP-2 and MMP-9 of human placental syncytiotrophoblast in vitro , which is not specific .

结论：丙型肝炎病毒持续感染时体外培养人胎盘合体滋养层细胞MMP-2和MMP-9合成分泌能力下降，为非特异性。

Conclusion : It shows that the reduced expression of Syndecan-1 in placenta syncytiotrophoblast of PIH may be one of the important factors responsible for the pathogenesis of PIH .

结论胎盘合体滋养层细胞Syndecan-1蛋白表达减少可能是妊高征的发病机制之一。

Computer image analysis ( 1 ) The expression of VEGF in placenta syncytiotrophoblast cells : There was no significant difference between mild PIH group and NP group , P > 0.05 ;

胎盘滋养细胞VEGF染色光度：轻度妊高征组和正常晚期妊娠组比较无显著差异，尸>0.05；中度妊高征组和轻度妊高征组比较无显著差异，尸>0.05；

The bulk of the human placenta consists of branching chorionic villi with an outer syncytiotrophoblast , underlying cytotrophoblasts , and a stromal core .

人胎盘绒毛膜绒毛的结构由内向外依次是绒毛基质，细胞滋养层细胞和其外层的合体细胞滋养细胞。

Results : Ability of invasion , adhesion , migration and function of hormone synthesis and secretion of infected syncytiotrophoblast decreased significantly in comparison with cells of control group ( P < 0.05 ) .

结果：感染组细胞侵袭、黏附、移动以及激素合成分泌功能较对照组均有显著下降（P<0.05）。

In pathological placentas , there are significantly lower expression of MMP-9 in syncytiotrophoblast ( 18.2 % ) than those without pathological change ( 75.0 % , P < 0.01 ) .

有病理改变的胎盘绒毛滋养细胞MMP-9的表达（18.2%）明显低于无病理改变胎盘（75.0%），差异有非常显著性（P<0.01）。

The results showed that the positive rate of neoplastic syncytiotrophoblast ( ST ) ; intermediate trophoblast ( IT ) and cytotrophoblast ( CT ) was 100 % respectively .

结果表明，肿瘤性合体滋养细胞（ST）、中间型滋养细胞（IT）及细胞滋养细胞（CT）内细胞角蛋白的阳性表达率均为100%。

Normal development of the placenta depends on the orchestrated balance of cytotrophoblast cell proliferation and differentiation into either one of the differentiated cell types , syncytiotrophoblast or invasive cytotrophoblasts .

胎盘的正常发育依赖于细胞滋养层细胞增殖动态平衡，和分化为合体滋养细胞和侵袭性细胞滋养层细胞的动态平衡。

In treatment group , the vacuoles in syncytiotrophoblast cell cytoplasms were reduced , the surface microvilli were increased and the endodermis cell organelles of the capillary were recovered .

治疗组合体滋养细胞胞浆空泡减少、表面微绒毛增加，毛细血管内皮细胞细胞器基本恢复正常。

Results IDO protein was expressed in the placenta , mainly in syncytiotrophoblast , of all the three groups , small amounts of IDO could be seen in vascular endothelial and interstitial cells .

结果各组孕产妇胎盘组织均有IDO蛋白表达，且主要表达于胎盘合体滋养细胞的胞质中，胎盘间质细胞及血管内皮细胞胞质中也可见少量表达。

Objective : To study the changes of the biochemical indexes in syncytiotrophoblast cell plasma membranes ( STCPM ) of pregnancy induced hypertension syndrome ( PIHS ) and the cause of these changes .

目的：研究妊高征（PIHS）患者胎盘合体滋养细胞质膜（STCPM）中生化指标的变化及原因。

The results demonstrated that apoptosis occurred in syncytiotrophoblast in rabbit pregnancy and apoptotic cell death increased from early to the end of gestation period , moreover Bax to Bcl-2 ratio may be related to apoptosis in placenta .

实验结果说明，在兔正常妊娠中，胎盘合体滋养层细胞发生凋亡，且随妊娠的进行，凋亡细胞数量增多，胎盘细胞凋亡主要与细胞中Bax∶Bcl2的比例相关。