Acute Gastric Mucosal Lesions

Experimental Study of Nimodipine in Prevention and Treatment of Acute Gastric Mucosal Lesions induced by Head Injury

尼莫地平防治颅脑伤后急性胃粘膜病变实验研究

Changes of gastric mucosal blood flow ( GMBF ) and potential difference ( PD ) in 50 healthy subjects and 45 patients with acute gastric mucosal lesions ( AGML ) Were studied by Laser Doppler flowmetry and PD measuring system under endoscopy .

应用激光多普勒血流计和胃粘膜电位直接测定法，在内镜下测定了50例正常对照者和45例急性胃粘膜病变患者的胃粘膜血流量（GMBF）和粘膜电位（PD）。

Early prevention and treatment of acute gastric mucosal lesions after abdominal operation

术后急性胃粘膜病变的早期防治

The Role of Calcium and cAMP in Acute Gastric Mucosal Lesions Induced by Indomethacin

钙及cAMP在消炎痛引起的急性胃粘膜损害中的作用

The importance of surgical treatment of massive upper gastrointestinal hemorrhage caused by acute gastric mucosal lesions

外科治疗在急性胃粘膜病变所致上消化道大出血治疗中的地位

The protective role of heat shock pretreatment on acute gastric mucosal lesions in rats caused by burn

热休克预处理对大鼠烧伤后急性胃黏膜损伤的保护作用

A clinical efficacy of octreotide in treatment of acute gastric mucosal lesions and stress ulcer with bleeding

奥曲肽治疗急性胃粘膜病变及应激性溃疡出血疗效观察

Relationship between acute gastric mucosal lesions and plasma β - endorphin after water-immersion stress in rats

大鼠浸水应激时胃粘膜损伤和β－内啡肽的关系

Changes of gastric acid production in early postburn period and their relevance to the prevention and treatment of acute gastric mucosal lesions

大鼠严重烧伤早期胃酸分泌功能改变的机制及其在急性胃粘膜损害防治中的意义

Conclusion Heat shock pretreatment is of protective role to acute gastric mucosal lesions in burned rats and the mechanism might be closely related to the enhanced expression of induced HSPs .

结论热休克预处理对烧伤大鼠胃黏膜具有保护作用，其保护机制可能与热休克蛋白诱导表达增强有关。

Objective : To establish a new animal mode of acute gastric mucosal lesions ( AGML ) in rats with open abdominal trauma after seawater immersion , so as to provide theoretical foundation for relevant study .

目的建立海水浸泡伤后急性胃黏膜病变（AGML）动物模型，为开展相关研究奠定理论基础。

Conclusion : Endogenous NO plays an important role in preventing stress induced acute gastric mucosal lesions by regulating GMBF . Inhibition or promotion of its synthesis can significantly exacerbate or mitigate acute gastric mucosal injuries .

结论：内源性NO对应激性胃粘膜损伤具有保护作用，抑制或促进其合成，可加重或减轻粘膜损伤，这一作用是通过影响粘膜血流量实现的。